1·CONCLUSION: ischemic postconditioning can relieve the ischemic reperfusion injury, the mechanism of which may be related with the decreased myocardial apoptosis.
结论：缺血后处理可减轻缺血再灌注损伤、其机制可能与减少心肌细胞凋亡有关。
2·There was no difference between benazepril postconditioning group and preconditioning group (P>0.05).
苯那普利后处理组和预处理组相比较，差异无统计学意义（P>0.05）。
3·Objective To investigate the protective effect of ischemia postconditioning on hepatic ischemia-reperfusion injury in rabbits.
目的探讨缺血后处理对肝脏热缺血再灌注损伤的保护作用。
4·But postconditioning is more controllable, then it have more clinical value. The mechanism of the protection, the time of clinical practice and the availability deserve deeply researching.
但缺血后处理具有更强的可控性，因而更具研究价值和临床实用意义，其器官保护机制、临床实施时机及其有效性，值得深入研究。
5·AIM: To investigate the protective effect of ischemic postconditioning on the functions of canine transplanted lung.
目的：观察缺血后处理对移植犬肺的呼吸功能的影响。